Cathepsin S-mediated fibroblast trans-differentiation contributes to left ventricular remodelling after myocardial infarction-Reference-Cited by-同舟云学术

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Cathepsin S-mediated fibroblast trans-differentiation contributes to left ventricular remodelling after myocardial infarction

Published:2013-06-14 Issue:1 Volume:100 Page:84-94
ISSN:1755-3245
Container-title:Cardiovascular Research
language:en
Short-container-title:

Author:

Chen Han,Wang Jing,Xiang Mei-Xiang,Lin Yan,He Aina,Jin Chun-Na,Guan Jian,Sukhova Galina K.,Libby Peter,Wang Jian-An,Shi Guo-Ping

Publisher

Oxford University Press (OUP)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology

Link

http://academic.oup.com/cardiovascres/article-pdf/100/1/84/17394404/cvt158.pdf

Reference40 articles.

1. Collagen network of the myocardium: function, structural remodeling and regulatory mechanisms;Weber;J Mol Cell Cardiol,1994

2. Fibronectin fragments containing the RGDS cell-binding domain mediate monocyte migration into the rabbit lung. A potential mechanism for C5 fragment-induced monocyte lung accumulation;Doherty;J Clin Invest,1990

3. Dualistic nature of adhesive protein function: fibronectin and its biologically active peptide fragments can autoinhibit fibronectin function;Yamada;J Cell Biol,1984

4. Atherosclerotic lesion development and Toll like receptor 2 and 4 responsiveness;Schoneveld;Atherosclerosis,2008

5. Lack of fibronectin-EDA promotes survival and prevents adverse remodeling and heart function deterioration after myocardial infarction;Arslan;Circ Res,2011

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