KCNJ5 mutations in familial and non-familial primary aldosteronism
Author:
Affiliation:
1. Department of Endocrinology-Diabetes Mellitus and Nutrition, Amiens University Hospital , 80054 Amiens , France
2. PériTox, UMR-I 01, University of Picardie Jules Verne , 80025 Amiens , France
Publisher
Oxford University Press (OUP)
Link
https://academic.oup.com/ejendo/advance-article-pdf/doi/10.1093/ejendo/lvae056/57846182/lvae056.pdf
Reference16 articles.
1. Therapeutic management of congenital forms of endocrine hypertension;Charoensri;Eur J Endocrinol,2023
2. K+ channel mutations in adrenal aldosterone-producing adenomas and hereditary hypertension;Choi;Science,2011
3. KCNJ5 mutations in European families with nonglucocorticoid remediable familial hyperaldosteronism;Mulatero;Hypertension,2012
4. Hypertension with or without adrenal hyperplasia due to different inherited mutations in the potassium channel KCNJ5;Scholl;Proc Natl Acad Sci U S A,2012
5. A case of severe hyperaldosteronism caused by a de novo mutation affecting a critical salt bridge Kir3.4 residue;Monticone;J Clin Endocrinol Metab,2015
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