Particulate threats: aryl hydrocarbon receptor, alveolar epithelium, environmentally persistent free radicals, and endothelial dysfunction-Reference-Cited by-同舟云学术

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Particulate threats: aryl hydrocarbon receptor, alveolar epithelium, environmentally persistent free radicals, and endothelial dysfunction

Published:2024-05-28 Issue:2 Volume:199 Page:161-162
ISSN:1096-6080
Container-title:Toxicological Sciences
language:en
Short-container-title:

Author:

Gowdy Kymberly M¹,Shannahan Jonathan²

Affiliation:

1. Department of Pulmonary, Critical Care and Sleep Medicine, Ohio State University Wexner Medical Center , Columbus, Ohio 43210, USA

2. School of Health Sciences, College of Health and Human Sciences, Purdue University , West Lafayette, Indiana 47907, USA

Publisher

Oxford University Press (OUP)

Link

https://academic.oup.com/toxsci/article-pdf/199/2/161/57890376/kfae036.pdf

Reference10 articles.

1. Source-dependent variation in hydroxyl radical production by airborne particulate matter;Alaghmand;Environ. Sci. Technol,2007

2. Inhalation of particulate matter containing environmentally persistent free radicals induces endothelial dysfunction mediated via AhR activation at the air-blood interface;Aryal;Toxicol. Sci,2024

3. Environmentally persistent free radicals: Methods for combustion generation, whole-body inhalation and assessing cardiopulmonary consequences;Aryal;Environ. Pollut,2023

4. Cardiovascular effects of particulate air pollution;Bhatnagar;Annu. Rev. Med,2022

5. Formation and stabilization of persistent free radicals;Dellinger;Proc. Combust. Inst,2007

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