Low-dose Bisphenol A ActivatesCyp11a1Gene Expression and Corticosterone Secretion in Adrenal Gland via the JNK Signaling Pathway
Author:
Publisher
Oxford University Press (OUP)
Subject
Toxicology
Link
http://academic.oup.com/toxsci/article-pdf/148/1/26/16693063/kfv162.pdf
Reference43 articles.
1. Inhibition of Testicular Steroidogenesis by the Xenoestrogen Bisphenol A Is Associated with Reduced Pituitary Luteinizing Hormone Secretion and Decreased Steroidogenic Enzyme Gene Expression in Rat Leydig Cells
2. Endocrine Disrupter Bisphenol A Increases In Situ Estrogen Production in the Mouse Urogenital Sinus
3. Low Doses of Bisphenol A Promote Human Seminoma Cell Proliferation by Activating PKA and PKG via a Membrane G-Protein–Coupled Estrogen Receptor
4. Xenoestrogen-Induced ERK-1 and ERK-2 Activation via Multiple Membrane-Initiated Signaling Pathways
5. Disconcordance in Statistical Models of Bisphenol A and Chronic Disease Outcomes in NHANES 2003-08
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