Secondhand Smoke-Prevalent Polycyclic Aromatic Hydrocarbon Binary Mixture-Induced Specific Mitogenic and Pro-inflammatory Cell Signaling Events in Lung Epithelial Cells

Author:

Osgood Ross S.1,Upham Brad L.2,Bushel Pierre R.3,Velmurugan Kalpana4,Xiong Ka-Na4,Bauer Alison K.4

Affiliation:

1. Department of Pharmaceutical Sciences, University of Colorado Anschutz Medical Campus, Aurora, Colorado 80045;

2. Department of Pediatrics and Human Development, Michigan State University, East Lansing, Michigan 48824;

3. Biostatistics & Computational Biology Branch, National Institute of Environmental Health Sciences, Research Triangle Park, North Carolina 27709;

4. Department of Environmental and Occupational Health, University of Colorado Anschutz Medical Center, Aurora, Colorado 80045

Funder

Flight Attendant Medical Research Institute

National Institutes of Health

Publisher

Oxford University Press (OUP)

Subject

Toxicology

Reference67 articles.

1. Heterocellular gap junctional communication between alveolar epithelial cells;Abraham;Am. J. Physiol. Lung Cell. Mol. Physiol.,2001

2. Amelioration of Benzo[a]pyrene-induced oxidative stress and pulmonary toxicity by Naringenin in Wistar rats: A plausible role of COX-2 and NF-kappaB;Ali;Hum. Exp. Toxicol.,2016

3. Increased susceptibility to urethane-induced lung tumors in mice with decreased expression of connexin43;Avanzo;Carcinogenesis,2004

4. Targeted deletion of Nrf2 reduces urethane-induced lung tumor development in mice;Bauer;PloS One,2011

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