Regulation of Nitrogen Mustard-Induced Lung Macrophage Activation by Valproic Acid, a Histone Deacetylase Inhibitor-Reference-Cited by-同舟云学术

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Regulation of Nitrogen Mustard-Induced Lung Macrophage Activation by Valproic Acid, a Histone Deacetylase Inhibitor

Published:2017-02-10 Issue:1 Volume:157 Page:222-234
ISSN:1096-6080
Container-title:Toxicological Sciences
language:en
Short-container-title:

Author:

Venosa Alessandro¹,Gow James G.¹,Hall LeRoy²,Malaviya Rama¹,Gow Andrew J.¹,Laskin Jeffrey D.³,Laskin Debra L.¹

Affiliation:

1. Department of Pharmacology and Toxicology, Rutgers University, Ernest Mario School of Pharmacy, Piscataway, New Jersey 08854;

2. Drug Safety Sciences, Johnson & Johnson, Raritan, New Jersey 08869;

3. Department of Environmental and Occupational Health, School of Public Health, Rutgers University, Piscataway, New Jersey 08854

Funder

National Institutes of Health

Publisher

Oxford University Press (OUP)

Subject

Toxicology

Link

http://academic.oup.com/toxsci/article-pdf/157/1/222/25423569/kfx032.pdf

Reference63 articles.

1. LPS regulates proinflammatory gene expression in macrophages by altering histone deacetylase expression;Aung;FASEB J,2006

2. Apolipoprotein E induces antiinflammatory phenotype in macrophages;Baitsch;Arterioscler. Thromb. Vasc. Biol.,2011

3. Apoptosis is a major pathway responsible for the resolution of type II pneumocytes in acute lung injury;Bardales;Am. J. Pathol.,1996

4. Ozone-induced lung inflammation and mucosal barrier disruption: Toxicology, mechanisms, and implications;Bhalla;J. Toxicol. Environ. Health B Crit. Rev.,1999

5. Histone deacetylase inhibitors decrease Toll-like receptor-mediated activation of proinflammatory gene expression by impairing transcription factor recruitment;Bode;Immunology,2007

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4. Suppression of Lung Oxidative Stress, Inflammation, and Fibrosis following Nitrogen Mustard Exposure by the Selective Farnesoid X Receptor Agonist Obeticholic Acid;Journal of Pharmacology and Experimental Therapeutics;2023-05-15

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