Role of Glucagon-Like Peptide-1 and Gastric Inhibitory Peptide in Anorexia Induction Following Oral Exposure to the Trichothecene Mycotoxin Deoxynivalenol (Vomitoxin)-Reference-Cited by-同舟云学术

Role of Glucagon-Like Peptide-1 and Gastric Inhibitory Peptide in Anorexia Induction Following Oral Exposure to the Trichothecene Mycotoxin Deoxynivalenol (Vomitoxin)

Published:2017-06-27 Issue:1 Volume:159 Page:16-24
ISSN:1096-6080
Container-title:Toxicological Sciences
language:en
Short-container-title:

Author:

Jia Hui,Wu Wen-Da,Lu Xi,Zhang Jie,He Cheng-Hua,Zhang Hai-Bin

Publisher

Oxford University Press (OUP)

Subject

Toxicology

Link

http://academic.oup.com/toxsci/article-pdf/159/1/16/19636539/kfx112.pdf

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1. Gut Microbiota Mediates Lactobacillus rhamnosus GG Alleviation of Deoxynivalenol-Induced Anorexia;Journal of Agricultural and Food Chemistry;2023-05-18

2. Neither glucagon-like peptide 1 receptors nor GDNF family receptor α-like neurons are required for aversive or anorectic response to deoxynivalenol (vomitoxin);American Journal of Physiology-Regulatory, Integrative and Comparative Physiology;2023-05-01

3. Substance P and Glucagon-like Peptide-17-36 Amide Mediate Anorexic Responses to Trichothecene Deoxynivalenol and Its Congeners;Toxins;2022-12-18

4. Emetic Response to T-2 Toxin Correspond to Secretion of Glucagon-like Peptide-17–36 Amide and Glucose-Dependent Insulinotropic Polypeptide;Toxins;2022-06-02

5. Glucose-Dependent Insulinotropic Polypeptide and Substance P Mediate Emetic Response Induction by Masked Trichothecene Deoxynivalenol-3-Glucoside through Ca2+ Signaling;Toxins;2022-05-27