All-trans Retinoic Acid Disrupts Development in Ex Vivo Cultured Fetal Rat Testes. II: Modulation of Mono-(2-ethylhexyl) Phthalate Toxicity

Author:

Spade Daniel J1ORCID,Hall Susan J1,Wortzel Jeremy D1,Reyes Gerardo12,Boekelheide Kim1

Affiliation:

1. Department of Pathology and Laboratory Medicine, Brown University, Providence, Rhode Island 02912

2. Division of Natural Sciences, College of Mount Saint Vincent, Riverdale, New York 10471

Funder

National Institute of Environmental Health Sciences

National Institute of General Medical Sciences

Eunice Kennedy Shriver

NICHD

NIH

Publisher

Oxford University Press (OUP)

Subject

Toxicology

Reference80 articles.

1. Absorption, metabolism, and excretion of di(2-ethylhexyl) phthalate by rats and mice;Albro;Environ. Health Perspect.,1986

2. A dose-response study following in utero and lactational exposure to di-(2-ethylhexyl) phthalate (DEHP): Effects on androgenic status, developmental landmarks and testicular histology in male offspring rats;Andrade;Toxicology,2006

3. Gene expression regulation by retinoic acid;Balmer;J. Lipid Res.,2002

4. Pathogenesis of male reproductive tract lesions from gestation through adulthood following in utero exposure to di(n-butyl) phthalate;Barlow;Toxicol. Pathol.,2003

5. Differential effects of phthalates on the testis and the liver;Bhattacharya;Biol. Reprod.,2005

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