Depletion of Securin Increases Arsenite-Induced Chromosome Instability and Apoptosis via a p53-Independent Pathway
Author:
Publisher
Oxford University Press (OUP)
Subject
Toxicology
Link
http://academic.oup.com/toxsci/article-pdf/90/1/73/16692396/kfj070.pdf
Reference64 articles.
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2. Amundson, S. A., Do, K. T., Vinikoor, L., Koch-Paiz, C. A., Bittner, M. L., Trent, J. M., Meltzer, P., and Fornace, A. J., Jr. (2005). Stress-specific signatures: Expression profiling of p53 wild-type and -null human cells. Oncogene24,4572–4579.
3. Bates, S., and Vousden, K. H. (1996). p53 in signaling checkpoint arrest or apoptosis. Curr. Opin. Genet. Dev.6,12–18.
4. Bernal, J. A., Luna, R., Espina, A., Lazaro, I., Ramos-Morales, F., Romero, F., Arias, C., Silva, A., Tortolero, M., and Pintor-Toro, J. A. (2002). Human securin interacts with p53 and modulates p53-mediated transcriptional activity and apoptosis. Nat. Genet.32,306–311.
5. Bhat, M. K., Yu, C., Yap, N., Zhan, Q., Hayashi, Y., Seth, P., and Cheng, S. (1997). Tumor suppressor p53 is a negative regulator in thyroid hormone receptor signaling pathways. J. Biol. Chem.272,28989–28993.
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