Involvement of Interleukin-6-Regulated Nitric Oxide Synthase in Hemorrhagic Cystitis and Impaired Bladder Contractions in Young Rats Induced by Acrolein, a Urinary Metabolite of Cyclophosphamide

Author:

Wang Ching-Chia,Weng Te-I,Wu En-Ting,Wu Mei-Hwan,Yang Rong-Sen,Liu Shing-Hwa

Publisher

Oxford University Press (OUP)

Subject

Toxicology

Reference37 articles.

1. Aminoguanidine, selective nitric oxide synthase inhibitor, ameliorates cyclophosphamide-induced hemorrhagic cystitis by inhibiting protein nitration and PARS activation;Urology,(2009)

2. Urinary excretion and pharmacokinetics of acrolein and its parent drug cyclophosphamide in bone marrow transplant patients;Bone Marrow Transplant.,(1998)

3. Preoperative plasma levels of interleukin-6 and its soluble receptor predict disease recurrence and survival of patients with bladder cancer;J. Urol.,(2002)

4. A model of hemorrhagic cystitis induced with acrolein in mice;Braz. J. Med. Biol. Res.,(2006)

5. Amifostine and glutathione prevent ifosfamide- and acrolein-induced hemorrhagic cystitis;Cancer Chemother. Pharmacol.,(2007)

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