Modulation of PC12 Cell Viability by Forskolin-Induced Cyclic AMP Levels Through ERK and JNK Pathways: An Implication for L-DOPA-Induced Cytotoxicity in Nigrostriatal Dopamine Neurons-Reference-Cited by-同舟云学术

Modulation of PC12 Cell Viability by Forskolin-Induced Cyclic AMP Levels Through ERK and JNK Pathways: An Implication for L-DOPA-Induced Cytotoxicity in Nigrostriatal Dopamine Neurons

Published:2012-04-26 Issue:1 Volume:128 Page:247-257
ISSN:1096-6080
Container-title:Toxicological Sciences
language:en
Short-container-title:Toxicological Sciences

Author:

Park K. H.,Park H. J.,Shin K. S.,Choi H. S.,Kai M.,Lee M. K.

Publisher

Oxford University Press (OUP)

Subject

Toxicology

Link

http://academic.oup.com/toxsci/article-pdf/128/1/247/10997455/kfs139.pdf

Reference50 articles.

1. l-DOPA Cytotoxicity to PC12 Cells in Culture Is via Its Autoxidation

2. Phosphorylation of Ser19 Alters the Conformation of Tyrosine Hydroxylase to Increase the Rate of Phosphorylation of Ser40

3. Induction of COX-2 and reactive gliosis by P2Y receptors in rat cortical astrocytes is dependent on ERK1/2 but independent of calcium signalling

4. Selective and persistent activation of extracellular signal-regulated protein kinase by nitric oxide in glial cells induces neuronal degeneration in glutathione-depleted midbrain cultures

5. Prostaglandin receptor EP2 protects dopaminergic neurons against 6-OHDA-mediated low oxidative stress

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