The Nrf2-Keap1-ARE Toxicity Pathway as a Cellular Sensor for Skin Sensitizers—Functional Relevance and a Hypothesis on Innate Reactions to Skin Sensitizers-Reference-Cited by-同舟云学术

The Nrf2-Keap1-ARE Toxicity Pathway as a Cellular Sensor for Skin Sensitizers—Functional Relevance and a Hypothesis on Innate Reactions to Skin Sensitizers

Published:2009-09-18 Issue:2 Volume:113 Page:284-292
ISSN:1096-6080
Container-title:Toxicological Sciences
language:en
Short-container-title:

Author:

Natsch Andreas

Publisher

Oxford University Press (OUP)

Subject

Toxicology

Link

http://academic.oup.com/toxsci/article-pdf/113/2/284/10939391/kfp228.pdf

Reference56 articles.

1. HMOX1 and NQO1 genes are upregulated in response to contact sensitizers in dendritic cells and THP-1 cell line: role of the Keap1/Nrf2 pathway;Toxicol. Sci.,2009

2. Reactivity profiling: covalent modification of single nucleophile peptides for skin sensitization risk assessment;Toxicol. Sci.,2009

3. Induction of phase II enzymes and hsp70 genes by copper sulfate through the electrophile-responsive element (EpRE): insights obtained from a transgenic zebrafish model carrying an orthologous EpRE sequence of mammalian origin;Fish Physiol. Biochem.,2009

4. IL-18 is a key proximal mediator of contact hypersensitivity and allergen-induced Langerhans cell migration in murine epidermis;J. Leukoc. Biol.,2008

5. Nonanimal alternatives for skin sensitization: a step forward?;Toxicol. Sci.,2008

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