Bromate-induced Changes in p21 DNA Methylation and Histone Acetylation in Renal Cells

Author:

Kolli Ramya T123,Glenn Travis C24,Brown Bradley T5,Kaur Sukhneeraj P1,Barnett Lillie M12,Lash Lawrence H6,Cummings Brian S12

Affiliation:

1. Department of Pharmaceutical and Biomedical Sciences

2. Interdisciplinary Toxicology Program

3. National Institute of Environmental Health Sciences, Building 101, 111 TW Alexander Drive, Durham, NC 27709

4. Environmental Health Science

5. College of Pharmacy, University of Georgia, Athens, Georgia 30602

6. Department of Pharmacology, Wayne State University, Detroit, Michigan 48201

Funder

National Institutes of Health

National Institute of Biomedical Imaging and Bioengineering

Interdisciplinary Toxicology Program Stipend Support

Publisher

Oxford University Press (OUP)

Subject

Toxicology

Reference60 articles.

1. HDAC inhibitors as anti-inflammatory agents;Adcock;Br. J. Pharmacol,2007

2. Basic local alignment search tool;Altschul;J. Mol. Biol,1990

3. P21WAF1/Cip1 gene is inactivated in metastatic prostatic cancer cell lines by promoter methylation;Bott;Prostate Cancer Prostatic Dis,2005

4. 5-Azacytidine induces transgene silencing by DNA methylation in Chinese hamster cells;Broday;Mol. Cell. Biol,1999

5. Up-regulation of p21(WAF1/Cip1) by histone deacetylase inhibitors reduces their cytotoxicity;Burgess;Mol. Pharmacol,2001

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