Pharmacologic activation of mitochondrial biogenesis exerts widespread beneficial effects in a transgenic mouse model of Huntington's disease-Reference-Cited by-同舟云学术

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Pharmacologic activation of mitochondrial biogenesis exerts widespread beneficial effects in a transgenic mouse model of Huntington's disease

Published:2011-11-17 Issue:5 Volume:21 Page:1124-1137
ISSN:1460-2083
Container-title:Human Molecular Genetics
language:en
Short-container-title:

Author:

Johri Ashu,Calingasan Noel Y.,Hennessey Thomas M.,Sharma Abhijeet,Yang Lichuan,Wille Elizabeth,Chandra Abhishek,Beal M. Flint

Publisher

Oxford University Press (OUP)

Subject

Genetics(clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/article-pdf/21/5/1124/17258641/ddr541.pdf

Reference60 articles.

1. Huntington disease;Vonsattel;J. Neuropathol. Exp. Neurol.,1998

2. Exon 1 of the HD gene with an expanded CAG repeat is sufficient to cause a progressive neurological phenotype in transgenic mice;Mangiarini;Cell,1996

3. Defects in adaptive energy metabolism with CNS-linked hyperactivity in PGC-1α null mice;Lin;Cell,2004

4. PGC-1α deficiency causes multi-system energy metabolic derangements: muscle dysfunction, abnormal weight control and hepatic steatosis;Leone;PLoS Biol.,2005

5. Suppression of reactive oxygen species and neurodegeneration by the PGC-1 transcriptional coactivators;St-Pierre;Cell,2006

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