Does loss of the normal protein function contribute to the pathogenesis of Huntington's disease?
Author:
Publisher
Oxford University Press (OUP)
Subject
General Agricultural and Biological Sciences,General Biochemistry, Genetics and Molecular Biology
Link
http://academic.oup.com/biohorizons/article-pdf/doi/10.1093/biohorizons/hzv005/4958641/hzv005.pdf
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3. Early Striatal Dendrite Deficits followed by Neuron Loss with Advanced Age in the Absence of Anterograde Cortical Brain-Derived Neurotrophic Factor
4. Intranuclear Neuronal Inclusions in Huntington's Disease and Dentatorubral and Pallidoluysian Atrophy: Correlation between the Density of Inclusions andIT15CAG Triplet Repeat Length
5. The toxic Aβ oligomer and Alzheimer's disease: an emperor in need of clothes
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1. Huntingtin CAG-expansion mutation results in a dominant negative effect;Frontiers in Cell and Developmental Biology;2023-09-01
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3. Zebrafish an experimental model of Huntington’s disease: molecular aspects, therapeutic targets and current challenges;Molecular Biology Reports;2021-10-19
4. Induced pluripotent stem cells as a potential treatment for Huntington’s disease;Recent Advances in iPSCs for Therapy, Volume 3;2021
5. Cell cycle re-entry of neurons and reactive neuroblastosis in Huntington's disease: Possibilities for neural-glial transition in the brain;Life Sciences;2020-12
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