Evidence for a role of PDZ domain-containing proteins to mediate hypophosphatemia in calcium stone formers
Author:
Affiliation:
1. Department of Medicine, University of Chicago, Chicago, IL, USA
2. Litholink Corporation, Laboratory Corporation of America Holdings, Chicago, IL, USA
Publisher
Oxford University Press (OUP)
Subject
Transplantation,Nephrology
Link
http://academic.oup.com/ndt/article-pdf/33/5/759/24652715/gfx284.pdf
Reference50 articles.
1. Hypophosphatemia and calcium nephrolithiasis;Prié;Nephron Exp Nephrol,2004
2. Frequency of renal phosphate leak among patients with calcium nephrolithiasis;Prié;Kidney Int,2001
3. A critical evaluation of the role of phosphate in the pathogenesis of absorptive hypercalciuria;Barilla;Miner Electrolyte Metabol,1979
4. A histomorphometric determination of iliac bone remodeling in patients with recurrent renal stone formation and idiopathic hypercalciuria;Steiniche;APMIS,1989
5. Effects of low-calcium diet on urine calcium excretion, parathyroid function and serum 1,25(OH)2D3 levels in patients with idiopathic hypercalciuria and in normal subjects;Coe;Am J Med,1982
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1. Evidence for abnormal linkage between urine oxalate and citrate excretion in human kidney stone formers;Physiological Reports;2021-07
2. Mechanisms for falling urine pH with age in stone formers;American Journal of Physiology-Renal Physiology;2019-07-01
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