COPI coatomer subunit α-COP interacts with the RNA binding protein Nucleolin via a C-terminal dilysine motif
Author:
Affiliation:
1. Dermatology, Indiana University School of Medicine , 545 Barnhill Drive, Emerson Hall 139, Indianapolis, IN 46202 , United States
2. Indiana University School of Medicine , 340 West 10 th St, Indianapolis, IN 46202, United States
Abstract
Funder
National Institute of Neurological Disorders and Stroke
Indiana University Medical Student Program for Research and Scholarship
National Institutes of Health
Publisher
Oxford University Press (OUP)
Subject
Genetics (clinical),Genetics,Molecular Biology,General Medicine
Link
https://academic.oup.com/hmg/advance-article-pdf/doi/10.1093/hmg/ddad140/51642994/ddad140.pdf
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3. The survival of motor neuron protein acts as a molecular chaperone for mRNP assembly;Donlin-Asp;Cell Rep,2017
4. SMN deficiency in severe models of spinal muscular atrophy causes widespread intron retention and DNA damage;Jangi;Proc Natl Acad Sci U S A,2017
5. Altered mRNA splicing in SMN-depleted motor neuron-like cells;Custer;PLoS One,2016
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