Novel forms of neurofascin 155 in the central nervous system: alterations in paranodal disruption models and multiple sclerosis-Reference-Cited by-同舟云学术

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Novel forms of neurofascin 155 in the central nervous system: alterations in paranodal disruption models and multiple sclerosis

Published:2010-02 Issue:2 Volume:133 Page:389-405
ISSN:1460-2156
Container-title:Brain
language:en
Short-container-title:

Author:

Pomicter Anthony D.,Shroff Seema M.,Fuss Babette,Sato-Bigbee Carmen,Brophy Peter J.,Rasband Matthew N.,Bhat Manzoor A.,Dupree Jeffrey L.

Publisher

Oxford University Press (OUP)

Subject

Clinical Neurology

Link

http://academic.oup.com/brain/article-pdf/133/2/389/16697649/awp341.pdf

Reference69 articles.

1. Neuronal membrane cholesterol loss enhances amyloid peptide generation;Abad-Rodriguez;J Cell Biol,2004

2. Two types of detergent-insoluble, glycosphingolipid/cholesterol-rich membrane domains from isolated myelin;Arvanitis;J Neurochem,2005

3. Lipid raft localization regulates the cleavage specificity of protease activated receptor 1 in endothelial cells;Bae;J Thromb Haemost,2008

4. Differential expression and functions of neuronal and glial neurofascin isoforms and splice variants during PNS development;Basak;Dev Biol,2007

5. Axon-glia interactions and the domain organization of myelinated axons requires neurexin IV/Caspr/Paranodin;Bhat;Neuron,2001

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