Tiam1 is part of a novel mechanism for morphine tolerance and hyperalgesia
Author:
Affiliation:
1. Center for Clinical Pharmacology, Department of Anesthesiology, Washington University School of Medicine , St. Louis, MO , USA
Abstract
Funder
US National Institutes of Health
Publisher
Oxford University Press (OUP)
Link
https://academic.oup.com/brain/advance-article-pdf/doi/10.1093/brain/awae170/58266963/awae170.pdf
Reference10 articles.
1. Differential opioid tolerance and opioid-induced hyperalgesia: A clinical reality;Hayhurst;Anesthesiology,2016
2. Tiam1-mediated maladaptive plasticity underlying morphine tolerance and hyperalgesia;Yao;Brain,2024
3. Control of synapse development and plasticity by Rho GTPase regulatory proteins;Tolias;Prog Neurobiol,2011
4. The Rac1-GEF Tiam1 couples the NMDA receptor to the activity-dependent development of dendritic arbors and spines;Tolias;Neuron,2005
5. Neuropathic pain memory is maintained by rac1-regulated dendritic spine remodeling after spinal cord injury;Tan;J Neurosci,2008
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