Restoring neuronal progranulin reverses deficits in a mouse model of frontotemporal dementia-Reference-Cited by-同舟云学术

Restoring neuronal progranulin reverses deficits in a mouse model of frontotemporal dementia

Published:2017-03-29 Issue:5 Volume:140 Page:1447-1465
ISSN:0006-8950
Container-title:Brain
language:en
Short-container-title:

Author:

Arrant Andrew E.¹,Filiano Anthony J.¹,Unger Daniel E.¹,Young Allen H.¹,Roberson Erik D.¹

Affiliation:

1. Center for Neurodegeneration and Experimental Therapeutics, Alzheimer’s Disease Center, and Evelyn F. McKnight Brain Institute, Departments of Neurology and Neurobiology, University of Alabama at Birmingham, Birmingham, AL, USA

Funder

National Institute of Neurological Disorders and Stroke

Glenn/AFAR Postdoctoral Fellowship

Kirschstein National Research Service Award Fellowship

Publisher

Oxford University Press (OUP)

Subject

Clinical Neurology

Link

http://academic.oup.com/brain/article-pdf/140/5/1447/25417405/awx060.pdf

Reference75 articles.

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2. Progranulin haploinsufficiency causes biphasic social dominance abnormalities in the tube test;Arrant;Genes Brain Behav,2016

3. Effects of exercise on progranulin levels and gliosis in progranulin-insufficient mice;Arrant;eNeuro,2015

4. Mutations in progranulin cause tau-negative FTD linked to chromosome 17;Baker;Nature,2006

5. Promoter DNA methylation regulates progranulin expression and is altered in FTLD;Banzhaf-Strathmann;Acta Neuropathol Commun,2013

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