Undirected compensatory plasticity contributes to neuronal dysfunction after severe spinal cord injury
Author:
Publisher
Oxford University Press (OUP)
Subject
Neurology (clinical)
Link
http://academic.oup.com/brain/article-pdf/136/11/3347/11137606/awt204.pdf
Reference69 articles.
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3. The injured spinal cord spontaneously forms a new intraspinal circuit in adult rats;Bareyre;Nat Neurosci,2004
4. Clonus after human spinal cord injury cannot be attributed solely to recurrent muscle-tendon stretch;Beres-Jones;Exp Brain Res,2003
5. Activation of 5-HT2A receptors upregulates the function of the neuronal K-Cl cotransporter KCC2;Bos;Proc Natl Acad Sci USA,2013
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