Nuclear trafficking in amyotrophic lateral sclerosis and frontotemporal lobar degeneration
Author:
Publisher
Oxford University Press (OUP)
Subject
Neurology (clinical)
Link
http://academic.oup.com/brain/article-pdf/140/1/13/10411312/aww197.pdf
Reference121 articles.
1. Cytoplasmic hGle1A regulates stress granules by modulation of translation
2. Aditi Glass L Dawson TR Wente SR . An amyotrophic lateral sclerosis-linked mutation in GLE1 alters the cellular pool of human Gle1 functional isoforms. Adv Biol Regul 2015b, in press. http://dx.doi.org/10.1016/j.jbior.2015.11.001 .
3. Inositol hexakisphosphate and Gle1 activate the DEAD-box protein Dbp5 for nuclear mRNA export
4. p62 positive, TDP-43 negative, neuronal cytoplasmic and intranuclear inclusions in the cerebellum and hippocampus define the pathology of C9orf72-linked FTLD and MND/ALS
5. Transcriptional down-regulation through nuclear exclusion of EWS methylated by PRMT1
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