Selective loss of cortical endothelial tight junction proteins during Alzheimer’s disease progression-Reference-Cited by-同舟云学术

Selective loss of cortical endothelial tight junction proteins during Alzheimer’s disease progression

Published:2019-02-15 Issue:4 Volume:142 Page:1077-1092
ISSN:0006-8950
Container-title:Brain
language:en
Short-container-title:

Author:

Yamazaki Yu¹,Shinohara Mitsuru¹,Shinohara Motoko¹,Yamazaki Akari¹,Murray Melissa E¹,Liesinger Amanda M¹,Heckman Michael G²,Lesser Elizabeth R²,Parisi Joseph E³⁴,Petersen Ronald C⁴,Dickson Dennis W¹,Kanekiyo Takahisa¹,Bu Guojun¹

Affiliation:

1. Department of Neuroscience, Mayo Clinic, Jacksonville, FL, USA

2. Division of Biomedical Statistics and Informatics, Mayo Clinic, Jacksonville, FL, USA

3. Department of Laboratory Medicine and Pathology, Mayo Clinic, Rochester, MN, USA

4. Department of Neurology, Mayo Clinic, Rochester, MN, USA

Funder

National Institutes of Health

NIH

Cure Alzheimer’s Fund

GHR Foundation

American Heart Association

Florida Department of Health Ed and Ethel Moore

Alzheimer’s Disease Research

Japan Society for the Promotion of Science

JSPS

Mochida Memorial Foundation for Medical & Pharmaceutical Research

Mayo Clinic Alzheimer's Disease Research Center and American Heart Association

Publisher

Oxford University Press (OUP)

Subject

Neurology (clinical)

Link

http://academic.oup.com/brain/article-pdf/142/4/1077/28229444/awz011.pdf

Reference68 articles.

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3. Focusing the amyloid cascade hypothesis on N-truncated Abeta peptides as drug targets against Alzheimer’s disease;Bayer;Acta Neuropathol,2014

4. Tau induces blood vessel abnormalities and angiogenesis-related gene expression in P301L transgenic mice and human Alzheimer’s disease;Bennett;Proc Natl Acad Sci USA,2018

5. Neuropathological stageing of Alzheimer-related changes;Braak;Acta Neuropathol,1991

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