Wild-type bone marrow cells ameliorate the phenotype of SOD1-G93A ALS mice and contribute to CNS, heart and skeletal muscle tissues-Reference-Cited by-同舟云学术

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Wild-type bone marrow cells ameliorate the phenotype of SOD1-G93A ALS mice and contribute to CNS, heart and skeletal muscle tissues

Published:2004-10-06 Issue:11 Volume:127 Page:2518-2532
ISSN:1460-2156
Container-title:Brain
language:en
Short-container-title:

Author:

Corti Stefania,Locatelli Federica,Donadoni Chiara,Guglieri Michela,Papadimitriou Dimitra,Strazzer Sandra,Del Bo Roberto,Comi Giacomo P.

Publisher

Oxford University Press (OUP)

Subject

Neurology (clinical)

Link

http://academic.oup.com/brain/article-pdf/127/11/2518/13733544/awh273.pdf

Reference37 articles.

1. Alvarez-Dolado M, Pardal R, Garcia-Verdugo JM, Fike JR, Lee HO, Pfeffer K, et al. Fusion of bone-marrow-derived cells with Purkinje neurons, cardiomyocytes and hepatocytes. Nature2003; 425: 968–73.

2. Asimakis GK, Lick S, Patterson C. Postischemic recovery of contractile function is impaired in SOD2(+/−) but not SOD1(+/−) mouse hearts. Circulation2002; 105: 981–6.

3. Azzouz M, Ralph GS, Storkebaum E, Walmsley LE, Mitrophanous KA, Kingsman SM, et al. VEGF delivery with retrogradely transported lentivector prolongs survival in a mouse ALS model. Nature2004; 429; 413–7.

4. Barlogie B, Shaughnessy J, Tricot G, Jacobson J, Zangari M, Anaissie E, et al. Treatment of multiple myeloma. Blood2004; 103: 20–32.

5. Beltrami AP, Urbanek K, Kajstura J, Yan SM, Finato N, Bussani R, et al. Evidence that human cardiac myocytes divide after myocardial infarction. N Engl J Med2001; 344: 1750–7.

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