Acute administration of tumour necrosis factor-α induces spontaneous calcium release via the reactive oxygen species pathway in atrial myocytes

Author:

Zuo Song1,Li Lin-ling1,Ruan Yan-fei1,Jiang Le1,Li Xin1,Li Song-nan1,Wen Song-nan1,Bai Rong1,Liu Nian1,Du Xin1,Dong Jian-zeng1,Ma Chang-Sheng1

Affiliation:

1. Department of Cardiology, Beijing Anzhen Hospital, Capital Medical University, National Clinical Research Center for Cardiovascular Diseases, No. 2 Anzhen Road, Chaoyang District, Beijing, China

Funder

National Natural Science Foundation of China

Beijing Natural Science Foundation

Publisher

Oxford University Press (OUP)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference20 articles.

1. Cellular and molecular electrophysiology of atrial fibrillation initiation, maintenance, and progression;Heijman;Circ Res,2014

2. Role of inflammation in atrial fibrillation pathophysiology and management;Harada;Circ J,2015

3. The effects of tumor necrosis factor-alpha on systolic and diastolic function in rat ventricular myocytes;Greensmith;Physiol Rep,2013

4. The role of Ca2+/calmodulin-dependent protein kinase II and calcineurin in TNF-alpha-induced myocardial hypertrophy;Wang;Braz J Med Biol Res,2012

5. Tumor necrosis factor-alpha decreases sarcoplasmic reticulum Ca2+-ATPase expressions via the promoter methylation in cardiomyocytes;Kao;Crit Care Med,2010

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