A genetic screen pinpoints ribonucleotide reductase residues that sustain dNTP homeostasis and specifies a highly mutagenic type of dNTP imbalance

Author:

Schmidt Tobias T12,Sharma Sushma3,Reyes Gloria X1,Gries Kerstin1,Gross Maike1,Zhao Boyu12,Yuan Jui-Hung4,Wade Rebecca456,Chabes Andrei37,Hombauer Hans1ORCID

Affiliation:

1. DNA Repair Mechanisms and Cancer, German Cancer Research Center (DKFZ), Heidelberg D-69120, Germany

2. Faculty of Bioscience, Heidelberg University, Heidelberg D-69120, Germany

3. Department of Medical Biochemistry and Biophysics, Umeå University, Umeå SE-901 87 Sweden

4. Molecular and Cellular Modeling Group, Heidelberg Institute for Theoretical Studies (HITS), Heidelberg D-69118, Germany

5. Interdisciplinary Center for Scientific Computing (IWR), Heidelberg D-69120, Germany

6. Center for Molecular Biology of the University of Heidelberg (ZMBH), DKFZ-ZMBH Alliance, Heidelberg D-69120, Germany

7. Laboratory for Molecular Infection Medicine Sweden, Umeå University, Umeå SE-901 87, Sweden

Funder

German Cancer Research Center

Swedish Cancer Society

Knut and Alice Wallenberg Foundation and the Swedish Research Council

Deutsches Krebsforschungszentrum

Publisher

Oxford University Press (OUP)

Subject

Genetics

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