Familial mesangio-capillary glomerulonephritis with initial presentation as haemolytic uraemic syndrome
Author:
Publisher
Oxford University Press (OUP)
Subject
Transplantation,Nephrology
Link
http://academic.oup.com/ndt/article-pdf/19/1/230/5131586/gfg470.pdf
Reference16 articles.
1. Hemolytic Uremic Syndrome in Families
2. Factor H Mutations in Hemolytic Uremic Syndrome Cluster in Exons 18–20, a Domain Important for Host Cell Recognition
3. Hypocomplementemia and Leukocytosis in Diarrhea-Associated Hemolytic Uremic Syndrome
4. Recurrent hemolytic-uremic syndrome with the hypomorphic fast allele of the third component of complement
5. Recurrent haemolytic uraemic syndrome and acquired hypomorphic variant of the third component of complement
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1. Functional Characterization of the Disease-Associated N-Terminal Complement Factor H Mutation W198R;Frontiers in Immunology;2017-12-13
2. Thrombotic Microangiopathy and the Kidney;Clinical Journal of the American Society of Nephrology;2017-10-17
3. Complement C5-inhibiting therapy for the thrombotic microangiopathies: accumulating evidence, but not a panacea;Clinical Kidney Journal;2017-05-08
4. Making sense of the spectrum of glomerular disease associated with complement dysregulation;Pediatric Nephrology;2013-07-14
5. Complement factor H mutation associated with membranoproliferative glomerulonephritis with transformation to atypical haemolytic uraemic syndrome;Clinical Kidney Journal;2013-02-05
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