Major basic protein-1 promotes fibrosis of dystrophic muscle and attenuates the cellular immune response in muscular dystrophy-Reference-Cited by-同舟云学术

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Major basic protein-1 promotes fibrosis of dystrophic muscle and attenuates the cellular immune response in muscular dystrophy

Published:2008-04-17 Issue:15 Volume:17 Page:2280-2292
ISSN:0964-6906
Container-title:Human Molecular Genetics
language:en
Short-container-title:Human Molecular Genetics

Author:

Wehling-Henricks M.,Sokolow S.,Lee J. J.,Myung K. H.,Villalta S. A.,Tidball J. G.

Publisher

Oxford University Press (OUP)

Subject

Genetics (clinical),Genetics,Molecular Biology,General Medicine

Reference53 articles.

1. Myonuclear apoptosis in dystrophic mdx muscle occurs by perforin-mediated cytotoxicity.

2. Helper (CD4+) and Cytotoxic (CD8+) T Cells Promote the Pathology of Dystrophin-Deficient Muscle

3. A nitric oxide synthase transgene ameliorates muscular dystrophy in mdx mice

4. Reduced necrosis of dystrophic muscle by depletion of host neutrophils, or blocking TNFα function with Etanercept in mdx mice

5. T-Cell-Dependent Fibrosis in the mdx Dystrophic Mouse

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