NCX is an important determinant for premature ventricular activity in a drug-induced model of Andersen

NCX is an important determinant for premature ventricular activity in a drug-induced model of Andersen–Tawil syndrome

Published:2011-06-21 Issue:1 Volume:92 Page:57-66
ISSN:1755-3245
Container-title:Cardiovascular Research
language:en
Short-container-title:

Author:

Radwański Przemysław B.¹²,Poelzing Steven¹³

Affiliation:

1. Nora Eccles Harrison Cardiovascular Research and Training Institute, University of Utah, 95 South 2000 East, Salt Lake City, UT 84112-5000, USA

2. Department of Pharmacology and Toxicology, University of Utah, 95 South 2000 East, Salt Lake City, UT 84112-5000, USA

3. Department of Bioengineering, University of Utah, 95 South 2000 East, Salt Lake City, UT 84112-5000, USA

Publisher

Oxford University Press (OUP)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology

Link

http://academic.oup.com/cardiovascres/article-pdf/92/1/57/25229341/cvr180.pdf

Reference28 articles.

1. Functional and clinical characterization of KCNJ2 mutations associated with LQT7 (Andersen syndrome);Tristani-Firouzi;J Clin Invest,2002

2. Mechanism of U wave and polymorphic ventricular tachycardia in a canine tissue model of Andersen–Tawil syndrome;Morita;Cardiovasc Res,2007

3. Relative importance of SR load and cytoplasmic calcium concentration in the genesis of aftercontractions in cardiac myocytes;Egdell;Cardiovasc Res,2000

4. The role of luminal Ca2+ in the generation of Ca2+ waves in rat ventricular myocytes;Lukyanenko;J Physiol,1999

5. Measurement of sarcoplasmic reticulum Ca2+ content and sarcolemmal Ca2+ fluxes in isolated rat ventricular myocytes during spontaneous Ca2+ release;Diaz;J Physiol,1997

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