Molecular mechanisms of activation of endothelial nitric oxide synthase mediated by transient receptor potential vanilloid type 1-Reference-Cited by-同舟云学术

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Molecular mechanisms of activation of endothelial nitric oxide synthase mediated by transient receptor potential vanilloid type 1

Published:2011-04-14 Issue:3 Volume:91 Page:492-501
ISSN:0008-6363
Container-title:Cardiovascular Research
language:en
Short-container-title:Cardiovascular Research

Author:

Ching L.-C.,Kou Y. R.,Shyue S.-K.,Su K.-H.,Wei J.,Cheng L.-C.,Yu Y.-B.,Pan C.-C.,Lee T.-S.

Publisher

Oxford University Press (OUP)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology

Link

http://academic.oup.com/cardiovascres/article-pdf/91/3/492/878711/cvr104.pdf

Reference34 articles.

1. Nitric oxide release accounts for the biological activity of endothelium-derived relaxing factor

2. Rho GTPases, Statins, and Nitric Oxide

3. Valsartan regulates the interaction of angiotensin II type 1 receptor and endothelial nitric oxide synthase via Src/PI3K/Akt signalling

4. Phosphorylation of Thr 495 Regulates Ca 2+ /Calmodulin-Dependent Endothelial Nitric Oxide Synthase Activity

5. Domain Mapping Studies Reveal That the M Domain of hsp90 Serves as a Molecular Scaffold to Regulate Akt-Dependent Phosphorylation of Endothelial Nitric Oxide Synthase and NO Release

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