Oral administration of cholera toxin B subunit conjugated to myelin basic protein protects against experimental autoimmune encephalomyelitis by inducing transforming growth factor-β-secreting cells and suppressing chemokine expression

Author:

Sun Jia-Bin,Xiao Bao-Guo,Lindblad Marianne,Li Bin-Ling,Link Hans,Czerkinsky Cecil,Holmgren Jan

Publisher

Oxford University Press (OUP)

Subject

Immunology,General Medicine,Immunology and Allergy

Reference36 articles.

1. Ortiz-Ortiz, L., Nakamura, R. M. and Weigle, W. O. 1976. T cell requirement for experimental allergic encephalomyelitis induction in the rat. J. Immunol.117:576.

2. Panitch, H. S. 1980. Adoptive transfer of experimental allergic encephalomyelitis with activated spleen cells: comparison on in vivo activation of concanvalin A. Cell. Immunol.56:1163.

3. Ben-Nun, A., Wekerle, H. and Cohen, I. 1981. The rapid isolation of clonal antigen specific lines capable of mediating autoimmune encephalomyelitis. Eur. J. Immunol.11:195.

4. Ben-Nun, A. and Cohen, I. R. 1982. Spontaneous remission and acquired resistance to autoimmune encephalomyelitis (EAE) are associated with suppression of T cell reactivity: suppressed EAE effector T cells recovered as T cell lines. J. Immunol.128:1450.

5. Bitar, D. M. and Whitacre, C. C. 1988. Suppression of experimental autoimmune encephalomyelitis by the oral administration of myelin basic protein. Cell. Immunol.112:364.

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