Persistently stalled replication forks inhibit nucleotide excision repair in trans by sequestering Replication protein A
Author:
Publisher
Oxford University Press (OUP)
Subject
Genetics
Link
http://academic.oup.com/nar/article-pdf/42/7/4406/
Reference27 articles.
1. Transcription-coupled nucleotide excision repair in mammalian cells: molecular mechanisms and biological effects;Fousteri;Cell Res.,2008
2. Eukaryotic nucleotide excision repair: from understanding mechanisms to influencing biology;Shuck;Cell Res.,2008
3. A dynamic model for replication protein A (RPA) function in DNA processing pathways;Fanning;Nucleic Acids Res,2006
4. Replication protein A safeguards genome integrity by controlling NER incision events;Overmeer;J. Cell Biol.,2011
5. Nucleotide excision repair is associated with the replisome and its efficiency depends on a direct interaction between XPA and PCNA;Gilljam;PloS One,2012
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4. A genome-wide screen reveals that Dyrk1A kinase promotes nucleotide excision repair by preventing aberrant co-stabilization of cyclin D1 and p21;2022-04-14
5. TREX1 degrades the 3′ end of the small DNA oligonucleotide products of nucleotide excision repair in human cells;Nucleic Acids Research;2022-03-31
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