Physiological and pathological functions of the prion protein homologue Dpl
Author:
Publisher
Oxford University Press (OUP)
Subject
General Medicine
Link
http://academic.oup.com/bmb/article-pdf/66/1/35/25152118/35.pdf
Reference30 articles.
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2. Bueler H, Fischer M, Lang Y et al. Normal development and behaviour of mice lacking the neuronal cell- surface PrP protein. Nature1992; 356: 577–82
3. Sakaguchi S, Katamine S, Nishida N et al. Loss of cerebellar Purkinje cells in aged mice homozygous for a disrupted PrP gene. Nature1996; 380: 528–31
4. Rossi D, Cozzio A, Flechsig E et al. Onset of ataxia and Purkinje cell loss in PrP null mice inversely correlated with Dpl level in brain. EMBO J2001; 20: 694–702
5. Moore RC, Lee IY, Silverman GL et al. Ataxia in prion protein (PrP)-deficient mice is associated with upregulation of the novel PrP-like protein Doppel [In Process Citation]. J Mol Biol1999; 292: 797–817
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