Novel role of calpain-3 in the triad-associated protein complex regulating calcium release in skeletal muscle-Reference-Cited by-同舟云学术

登录注册会员服务联系我们

Novel role of calpain-3 in the triad-associated protein complex regulating calcium release in skeletal muscle

Published:2008-11-01 Issue:21 Volume:17 Page:3271-3280
ISSN:0964-6906
Container-title:Human Molecular Genetics
language:en
Short-container-title:Human Molecular Genetics

Author:

Kramerova I.,Kudryashova E.,Wu B.,Ottenheijm C.,Granzier H.,Spencer M. J.

Publisher

Oxford University Press (OUP)

Subject

Genetics(clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/article-pdf/17/21/3271/7069178/ddn223.pdf

Reference41 articles.

1. Molecular and cellular basis of calpainopathy (limb girdle muscular dystrophy type 2A)

2. A third of LGMD2A biopsies have normal calpain 3 proteolytic activity as determined by an in vitro assay

3. Null mutation of calpain 3 (p94) in mice causes abnormal sarcomere formation in vivo and in vitro

4. Muscle-specific Calpain, p94, Responsible for Limb Girdle Muscular Dystrophy Type 2A, Associates with Connectin through IS2, a p94-specific Sequence

5. Muscle-Specific Calpain, p94, Interacts with the Extreme C-Terminal Region of Connectin, a Unique Region Flanked by Two Immunoglobulin C2 Motifs

Cited by 85 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Loss of Calpain 3 dysregulates store‐operated calcium entry and its exercise response in mice;The FASEB Journal;2024-07-19

2. Human calpain-3 and its structural plasticity: dissociation of a homohexamer into dimers on binding titin;2024-03-03

3. A knock down strategy for rapid, generic, and versatile modelling of muscular dystrophies in 3D-tissue-engineered-skeletal muscle;Skeletal Muscle;2024-02-22

4. α-Glucan derivatives as selective blockers of aldolase A: Computer-aided structure optimization and the effects on HCC;Carbohydrate Polymers;2024-02

5. Loss of calpain 3 dysregulates store-operated calcium entry and its exercise response in mice;2024-01-15

同舟云学术

1.学者识别学者识别

2.学术分析学术分析

3.人才评估人才评估

"同舟云学术"是以全球学者为主线，采集、加工和组织学术论文而形成的新型学术文献查询和分析系统，可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容，当前同舟云学术共收录了国内外主流学术期刊6万余种，收集的期刊论文及会议论文总量共计约1.5亿篇，并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询！咨询电话：010-8811{复制后删除}0370

www.globalauthorid.com

TOP

Copyright © 2019-2024 北京同舟云网络信息技术有限公司
京公网安备11010802033243号京ICP备18003416号-3