Complement C5a inhibition moderates lipid metabolism and reduces tubulointerstitial fibrosis in diabetic nephropathy
Author:
Affiliation:
1. Division of Nephrology, Department of Medicine, Queen Mary Hospital, University of Hong Kong, Pokfulam, Hong Kong
2. Medical Research Council Centre for Transplantation, King’s College London, Guy’s Hospital, London, UK
Funder
Hong Kong Society of Nephrology Research
Medical Research Council of the UK
Guangdong Medical University
Yu Professorship in Nephrology
University of Hong Kong
Winston Leung K. K. Chan of the Hong Kong Concrete Co.
L & T Charitable Foundation Ltd. & Indo Café
Publisher
Oxford University Press (OUP)
Subject
Transplantation,Nephrology
Link
http://academic.oup.com/ndt/article-pdf/33/8/1323/25198340/gfx336.pdf
Reference43 articles.
1. Diabetic kidney disease: act now or pay later;Atkins;Med J Aust,2010
2. Tubulointerstitial changes as a major determinant in the progression of renal damage;Nath;Am J Kidney Dis,1992
3. Anti-fibrosis therapy and diabetic nephropathy;Karihaloo;Curr Diab Rep,2012
4. Molecular targets for treatment of kidney fibrosis;Chuang;J Mol Med,2013
5. Complement activation in progressive renal disease;Fearn;World J Nephrol,2015
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