Diet and Cancer Prevention Studies in p53-Deficient Mice
Author:
Affiliation:
1. National Cancer Institute, Bethesda, MD 20892
Publisher
Oxford University Press (OUP)
Subject
Nutrition and Dietetics,Medicine (miscellaneous)
Link
http://academic.oup.com/jn/article-pdf/131/11/3092S/24003244/4w11u1s3092.pdf
Reference26 articles.
1. Mechanism-based cancer prevention approaches: targets, examples and the use of transgenic mice;Hursting;J. Natl. Cancer Inst.,1999
2. p53 mutations in human cancers;Hollstein;Science (Washington DC),1991
3. Mice deficient for p53 are developmentally normal but susceptible to spontaneous tumors;Donehower;Nature (Lond.),1992
4. Reduction of p53 gene dosage does not increase initiation or promotion but enhances malignant progression of chemically induced skin tumors;Kemp;Cell,1993
5. Calorie restriction delays spontaneous tumorigenesis in p53-knockout transgenic mice;Hursting;Proc. Natl. Acad. Sci. U.S.A.,1994
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