The lysosomal inhibitor, chloroquine, increases cell surface BMPR-II levels and restores BMP9 signalling in endothelial cells harbouring BMPR-II mutations-Reference-Cited by-同舟云学术

The lysosomal inhibitor, chloroquine, increases cell surface BMPR-II levels and restores BMP9 signalling in endothelial cells harbouring BMPR-II mutations

Published:2013-05-12 Issue:18 Volume:22 Page:3667-3679
ISSN:0964-6906
Container-title:Human Molecular Genetics
language:en
Short-container-title:Human Molecular Genetics

Author:

Dunmore B. J.,Drake K. M.,Upton P. D.,Toshner M. R.,Aldred M. A.,Morrell N. W.

Publisher

Oxford University Press (OUP)

Subject

Genetics(clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/article-pdf/22/18/3667/17259594/ddt216.pdf

Reference53 articles.

1. Cellular and Molecular Basis of Pulmonary Arterial Hypertension

2. Pulmonary Hypertension Due to BMPR2 Mutation: A New Paradigm for Tissue Remodeling?

3. Heterozygous germline mutations in BMPR2, encoding a TGF-β receptor, cause familial primary pulmonary hypertension

4. BMPR2 Haploinsufficiency as the Inherited Molecular Mechanism for Primary Pulmonary Hypertension

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