Endocytosis machinery is required for β1-adrenergic receptor-induced hypertrophy in neonatal rat cardiac myocytes-Reference-Cited by-同舟云学术

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Endocytosis machinery is required for β1-adrenergic receptor-induced hypertrophy in neonatal rat cardiac myocytes

Published:2008-01-14 Issue:1 Volume:78 Page:36-44
ISSN:1755-3245
Container-title:Cardiovascular Research
language:en
Short-container-title:

Author:

Morisco Carmine,Marrone Chiara,Galeotti Jonathan,Shao Dan,Vatner Dorothy E.,Vatner Stephen F.,Sadoshima Junichi

Publisher

Oxford University Press (OUP)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology

Link

http://academic.oup.com/cardiovascres/article-pdf/78/1/36/17197514/cvn008.pdf

Reference34 articles.

1. Beta-adrenergic receptor signaling: an acute compensatory adjustment—inappropriate for the chronic stress of heart failure? Insights from Gsalpha overexpression and other genetically engineered animal models;Vatner;Circ Res,2000

2. Altered beta-adrenergic receptor gene regulation and signaling in chronic heart failure;Port;J Mol Cell Cardiol,2001

3. Augmenting beta receptors in the heart: short-term gains offset by long-term pains?;Limbird;Proc Natl Acad Sci USA,1999

4. Beta-adrenergic cardiac hypertrophy is mediated primarily by the beta(1)-subtype in the rat heart;Morisco;J Mol Cell Cardiol,2001

5. Progressive hypertrophy and heart failure in beta1-adrenergic receptor transgenic mice;Engelhardt;Proc Natl Acad Sci USA,1999

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