Resistance of bulky DNA lesions to nucleotide excision repair can result from extensive aromatic lesion–base stacking interactions-Reference-Cited by-同舟云学术

Resistance of bulky DNA lesions to nucleotide excision repair can result from extensive aromatic lesion–base stacking interactions

Published:2011-07-15 Issue:20 Volume:39 Page:8752-8764
ISSN:1362-4962
Container-title:Nucleic Acids Research
language:en
Short-container-title:

Author:

Reeves Dara A.,Mu Hong,Kropachev Konstantin,Cai Yuqin,Ding Shuang,Kolbanovskiy Alexander,Kolbanovskiy Marina,Chen Ying,Krzeminski Jacek,Amin Shantu,Patel Dinshaw J.,Broyde Suse,Geacintov Nicholas E.

Publisher

Oxford University Press (OUP)

Subject

Genetics

Link

http://academic.oup.com/nar/article-pdf/39/20/8752/16777925/gkr537.pdf

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3. Molecular mechanisms of mammalian global genome nucleotide excision repair;Gillet;Chem. Rev.,2006

4. The human DNA repair factor XPC-HR23B distinguishes stereoisomeric benzo[ a ]pyrenyl-DNA lesions;Mocquet;EMBO J.,2007

5. Sequential recruitment of the repair factors during NER: the role of XPG in initiating the resynthesis step;Mocquet;EMBO J.,2008

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