Macrophage p38 Mitogen-Activated Protein Kinase Activity Regulates Invariant Natural Killer T-Cell Responses During Borrelia burgdorferi Infection

Author:

Hawley Kelly,Navasa Nicolás,Olson Chris M.,Bates Tonya C.,Garg Renu,Hedrick Michael N.,Conze Dietrich,Rincón Mercedes,Anguita Juan

Publisher

Oxford University Press (OUP)

Subject

Infectious Diseases,Immunology and Allergy

Reference48 articles.

1. Toll-like receptor 2 is required for innate, but not acquired, host defense to Borrelia burgdorferi;Wooten;J Immunol,2002

2. Identification of a TLR-independent pathway for Borrelia burgdorferi-induced expression of matrix metalloproteinases and inflammatory mediators through binding to integrin α3β1;Behera;J Immunol,2006

3. Cutting edge: inflammatory signaling by Borrelia burgdorferi lipoproteins is mediated by toll-like receptor 2;Hirschfeld;J Immunol,1999

4. Borrelia burgdorferi activates nuclear factor-kappa B and is a potent inducer of chemokine and adhesion molecule gene expression in endothelial cells and fibroblasts;Ebnet;J Immunol,1997

5. Murine Lyme arthritis development mediated by p38 mitogen-activated protein kinase activity;Anguita;J Immunol,2002

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