Soluble Aβ Oligomers Formed Channels Leading to Calcium Dysregulation
Author:
Affiliation:
1. Ann Romney Center for Neurologic Diseases, Brigham and Women’s Hospital and Harvard Medical School , Boston, MA 02115 , USA
Publisher
Oxford University Press (OUP)
Subject
Cancer Research,Cell Biology,Molecular Medicine,Physiology
Link
https://academic.oup.com/function/advance-article-pdf/doi/10.1093/function/zqad037/50898142/zqad037.pdf
Reference9 articles.
1. Reversal of calcium dysregulation as potential approach for treating Alzheimer’s disease;Popugaeva;Curr Alzheimer Res,2020
2. Calcium dyshomeostasis in Alzheimer’s Disease pathogenesis;Cascella;Int J Mol Sci,2021
3. Endogenous amyloid-formed Ca2+-permeable channels in aged 3xTg AD mice;Li;Function (Oxf),2023
4. A mechanistic hypothesis for the impairment of synaptic plasticity by soluble aβ oligomers from Alzheimer’s brain;Li;J Neurochem,2020
5. The amyloid-β oligomer hypothesis: beginning of the third decade;Cline;J Alzheimers Dis,2018
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