KATP Channel Function: More than Meets the Eye
Author:
Affiliation:
1. Department of Chemical Physiology and Biochemistry, School of Medicine, Oregon Health and Science University, Portland OR 97225, USA
Funder
National Institutes of Health
Publisher
Oxford University Press (OUP)
Subject
General Medicine
Link
https://academic.oup.com/function/advance-article-pdf/doi/10.1093/function/zqab070/42124667/zqab070.pdf
Reference10 articles.
1. Molecular biology of adenosine triphosphate-sensitive potassium channels;Aguilar-Bryan;Endocr Rev,1999
2. Activating mutations in the gene encoding the ATP-sensitive potassium-channel subunit Kir6.2 and permanent neonatal diabetes;Gloyn;N Engl J Med,2004
3. Targeted overactivity of beta cell K(ATP) channels induces profound neonatal diabetes;Koster;Cell,2000
4. Activating mutations in Kir6.2 and neonatal diabetes: new clinical syndromes, new scientific insights, and new therapy;Hattersley;Diabetes,2005
5. Muscle dysfunction caused by a KATP channel mutation in neonatal diabetes is neuronal in origin;Clark;Science,2010
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