Pitx2c increases in atrial myocytes from chronic atrial fibrillation patients enhancingIKsand decreasingICa,L-Reference-Cited by-同舟云学术

Pitx2c increases in atrial myocytes from chronic atrial fibrillation patients enhancingIKsand decreasingICa,L

Published:2015-12-28 Issue:3 Volume:109 Page:431-441
ISSN:0008-6363
Container-title:Cardiovascular Research
language:en
Short-container-title:Cardiovasc Res

Author:

Pérez-Hernández Marta,Matamoros Marcos,Barana Adriana,Amorós Irene,Gómez Ricardo,Núñez Mercedes,Sacristán Sandra,Pinto Ángel,Fernández-Avilés Francisco,Tamargo Juan,Delpón Eva,Caballero Ricardo

Funder

Centro Nacional de Investigaciones Cardiovasculares

Ministerio de Ciencia e Innovación

Instituto de Salud Carlos III

Red Investigación Cardiovascular

Comunidad Autónoma de Madrid

Fundaciones Mutua Madrileña and BBVA Grants

Publisher

Oxford University Press (OUP)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology

Link

http://academic.oup.com/cardiovascres/article-pdf/109/3/431/17195748/cvv280.pdf

Reference34 articles.

1. An update on atrial fibrillation in 2014: from pathophysiology to treatment;Ferrari;Int J Cardiol,2015

2. The impact of new and emerging clinical data on treatment strategies for atrial fibrillation;Prystowsky;J Cardiovasc Electrophysiol,2010

3. Atrial Remodeling and Atrial Fibrillation

4. Atrial L-Type Ca 2+ Currents and Human Atrial Fibrillation

5. In Humans, Chronic Atrial Fibrillation Decreases the Transient Outward Current and Ultrarapid Component of the Delayed Rectifier Current Differentially on Each Atria and Increases the Slow Component of the Delayed Rectifier Current in Both

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