Loss of positive allosteric interactions between neuronal nitric oxide synthase and phosphofructokinase contributes to defects in glycolysis and increased fatigability in muscular dystrophy-Reference-Cited by-同舟云学术

Loss of positive allosteric interactions between neuronal nitric oxide synthase and phosphofructokinase contributes to defects in glycolysis and increased fatigability in muscular dystrophy

Published:2009-06-19 Issue:18 Volume:18 Page:3439-3451
ISSN:0964-6906
Container-title:Human Molecular Genetics
language:en
Short-container-title:Human Molecular Genetics

Author:

Wehling-Henricks M.,Oltmann M.,Rinaldi C.,Myung K. H.,Tidball J. G.

Publisher

Oxford University Press (OUP)

Subject

Genetics(clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/article-pdf/18/18/3439/1473001/ddp288.pdf

Reference58 articles.

1. Nitric oxide synthase complexed with dystrophin and absent from skeletal muscle sarcolemma in Duchenne muscular dystrophy

2. Neuronal nitric oxide synthase and dystrophin-deficient muscular dystrophy.

3. Impaired metabolic modulation of -adrenergic vasoconstriction in dystrophin-deficient skeletal muscle

4. Functional muscle ischemia in neuronal nitric oxide synthase-deficient skeletal muscle of children with Duchenne muscular dystrophy

5. A nitric oxide synthase transgene ameliorates muscular dystrophy in mdx mice

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