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Defective pulmonary vascular remodeling in Smad8 mutant mice

Published:2009-05-05 Issue:15 Volume:18 Page:2791-2801
ISSN:0964-6906
Container-title:Human Molecular Genetics
language:en
Short-container-title:Human Molecular Genetics

Author:

Huang Z.,Wang D.,Ihida-Stansbury K.,Jones P. L.,Martin J. F.

Publisher

Oxford University Press (OUP)

Subject

Genetics (clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/article-pdf/18/15/2791/1685438/ddp214.pdf

Reference65 articles.

1. Bone morphogenetic proteins: multifunctional regulators of vertebrate development.

2. Smad-dependent and Smad-independent pathways in TGF-β family signalling

3. SMAD proteins control DROSHA-mediated microRNA maturation

4. From developmental disorder to heritable cancer: it's all in the BMP/TGF-β family

5. To be or not to BMP

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1. Integrative genomic analyses identify candidate causal genes for calcific aortic valve stenosis involving tissue-specific regulation;Nature Communications;2024-03-18

2. The evaluation of the SMAD1 rs1016792 polymorphism and gene expression on pulmonary hypertension due to congenital heart disease in children: a preliminary study;Nucleosides, Nucleotides & Nucleic Acids;2024-02-26

3. Bone Substitute Options for Spine Fusion in Patients With Spine Trauma-Part II: The Role of rhBMP;Korean Journal of Neurotrauma;2024

4. Defining the clinical validity of genes reported to cause pulmonary arterial hypertension;Genetics in Medicine;2023-11

5. Pulmonary arterial hypertension drugs can partially restore altered angiogenic capacities in bmpr2‐silenced human lung microvascular endothelial cells;Pulmonary Circulation;2023-10

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