The pro-apoptotic JNK scaffold POSH/SH3RF1 mediates CHMP2BIntron5-associated toxicity in animal models of frontotemporal dementia
Author:
Affiliation:
1. Department of Biology, University of York, York YO10 5DD, UK
2. Department of Neurology, University of Massachusetts Medical School, Worcester, MA 01605, USA
Funder
Medical Research Council
National Institutes of Health
Publisher
Oxford University Press (OUP)
Subject
Genetics(clinical),Genetics,Molecular Biology,General Medicine
Link
http://academic.oup.com/hmg/article-pdf/27/8/1382/24527075/ddy048.pdf
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3. Frontotemporal dementia and its subtypes: a genome-wide association study;Ferrari;Lancet Neurol,2014
4. Progranulin deficiency promotes circuit-specific synaptic pruning by microglia via complement activation;Lui;Cell,2016
5. Increased intrathecal inflammatory activity in frontotemporal dementia: pathophysiological implications;Sjogren;J. Neurol. Neurosurg. Psychiatry,2004
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