Deactivation of TBP contributes to SCA17 pathogenesis-Reference-Cited by-同舟云学术

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Deactivation of TBP contributes to SCA17 pathogenesis

Published:2014-08-07 Issue:25 Volume:23 Page:6878-6893
ISSN:1460-2083
Container-title:Human Molecular Genetics
language:en
Short-container-title:

Author:

Hsu Tun-Chieh,Wang Cheng-Kuang,Yang Chun-Yen,Lee Li-Ching,Hsieh-Li Hsiu-Mei,Ro Long-Sun,Chen Chiung-Mei,Lee-Chen Guey-Jen,Su Ming-Tsan

Publisher

Oxford University Press (OUP)

Subject

Genetics (clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/article-pdf/23/25/6878/13916773/ddu410.pdf

Reference44 articles.

1. Molecular pathogenesis of spinocerebellar ataxias;Duenas;Brain,2006

2. Polyglutamine diseases: emerging concepts in pathogenesis and therapy;Shao;Hum. Mol. Genet.,2007

3. Detection of polyglutamine protein oligomers in cells by fluorescence correlation spectroscopy;Takahashi;J. Biol. Chem.,2007

4. Cell death triggered by polyglutamine-expanded huntingtin in a neuronal cell line is associated with degradation of CREB-binding protein;Jiang;Hum. Mol. Genet.,2003

5. Recruitment and the role of nuclear localization in polyglutamine-mediated aggregation;Perez;J. Cell. Biol.,1998

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