Silencing of either SR-A or CD36 reduces atherosclerosis in hyperlipidaemic mice and reveals reciprocal upregulation of these receptors-Reference-Cited by-同舟云学术

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Silencing of either SR-A or CD36 reduces atherosclerosis in hyperlipidaemic mice and reveals reciprocal upregulation of these receptors

Published:2010-07-15 Issue:3 Volume:88 Page:530-538
ISSN:0008-6363
Container-title:Cardiovascular Research
language:en
Short-container-title:Cardiovascular Research

Author:

Makinen P. I.,Lappalainen J. P.,Heinonen S. E.,Leppanen P.,Lahteenvuo M. T.,Aarnio J. V.,Heikkila J.,Turunen M. P.,Yla-Herttuala S.

Publisher

Oxford University Press (OUP)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine,Physiology

Link

http://academic.oup.com/cardiovascres/article-pdf/88/3/530/1132365/cvq235.pdf

Reference36 articles.

1. The Response-to-Retention Hypothesis of Early Atherogenesis

2. Glycosaminoglycans and apolipoproteins B and A-I in human aortas. Chemical and immunological analysis of lesion-free aortas from children and adults.

3. Beyond Cholesterol

4. The macrophage foam cell as a target for therapeutic intervention

5. Gene expression in macrophage-rich human atherosclerotic lesions. 15-lipoxygenase and acetyl low density lipoprotein receptor messenger RNA colocalize with oxidation specific lipid-protein adducts.

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