The small heat shock protein B8 (HspB8) promotes autophagic removal of misfolded proteins involved in amyotrophic lateral sclerosis (ALS)-Reference-Cited by-同舟云学术

The small heat shock protein B8 (HspB8) promotes autophagic removal of misfolded proteins involved in amyotrophic lateral sclerosis (ALS)

Published:2010-06-22 Issue:17 Volume:19 Page:3440-3456
ISSN:1460-2083
Container-title:Human Molecular Genetics
language:en
Short-container-title:

Author:

Crippa Valeria,Sau Daniela,Rusmini Paola,Boncoraglio Alessandra,Onesto Elisa,Bolzoni Elena,Galbiati Mariarita,Fontana Elena,Marino Marianna,Carra Serena,Bendotti Caterina,De Biasi Silvia,Poletti Angelo

Publisher

Oxford University Press (OUP)

Subject

Genetics (clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/article-pdf/19/17/3440/13905489/ddq257.pdf

Reference51 articles.

1. Lessons from models of SOD1-linked familial ALS;Bendotti;Trends Mol. Med.,2004

2. Ubiquitinated TDP-43 in frontotemporal lobar degeneration and amyotrophic lateral sclerosis;Neumann;Science,2006

3. Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis;Rosen;Nature,1993

4. TARDBP mutations in individuals with sporadic and familial amyotrophic lateral sclerosis;Kabashi;Nat. Genet.,2008

5. TDP-43 mutations in familial and sporadic amyotrophic lateral sclerosis;Sreedharan;Science,2008

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