Redox homeostasis, T cells and kidney diseases: three faces in the dark-Reference-Cited by-同舟云学术

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Redox homeostasis, T cells and kidney diseases: three faces in the dark

Published:2015-12-13 Issue:1 Volume:9 Page:1-10
ISSN:2048-8505
Container-title:Clinical Kidney Journal
language:en
Short-container-title:Clin Kidney J

Author:

Simeoni Luca,Thurm Christoph,Kritikos Andreas,Linkermann Andreas

Funder

German Research Foundation (DFG)

Cluster of Excellence ‘Inflammation at Interfaces’

Publisher

Oxford University Press (OUP)

Subject

Transplantation,Nephrology

Link

http://academic.oup.com/ckj/article-pdf/9/1/1/7451556/sfv135.pdf

Reference119 articles.

1. Redox Regulation of Inflammation: Old Elements, a New Story

2. Ferroptosis: An Iron-Dependent Form of Nonapoptotic Cell Death

3. Synchronized renal tubular cell death involves ferroptosis

4. Regulated necrosis: the expanding network of non-apoptotic cell death pathways

5. T Cell Activation

Cited by 9 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Association of redox and inflammation-related biomarkers with prognosis in IgA nephropathy: A prospective observational study;Free Radical Biology and Medicine;2022-08

2. Mesenchymal stromal cell–based therapies for acute kidney injury: progress in the last decade;Kidney International;2020-06

3. Genetic disruption of Npr1 depletes regulatory T cells and provokes high levels of proinflammatory cytokines and fibrosis in the kidneys of female mutant mice;American Journal of Physiology-Renal Physiology;2019-06-01

4. Glutathione de novo synthesis but not recycling process coordinates with glutamine catabolism to control redox homeostasis and directs murine T cell differentiation;eLife;2018-09-10

5. Impaired X-CGD T cell compartment is gp91phox-NADPH oxidase independent;Clinical Immunology;2018-08

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